SLC52A2 and renal tubular dysgenesis of genetic origin: Meanwhile, following gene therapy with the AAV9-SLC52A2 vector the neural network in RTD patient was preserved over time, showing a progressive amelioration of the neurites’ length in transduced RTD motoneurons (Figure 5C, **p = 0.001, ANOVA; mean ± SEM; N = 3 independent experiments, N = 4 images per independent experiment per sample).