FGF23 and hypophosphatemia: The role of FGF23 fragments is not fully understood yet, though c-term FGF23 can compete with the intact molecule for its binding to FGFR, acting as an endogenous inhibitor of αKlotho-FGFR complex formation and subsequent intact FGF23 signaling, which may thus reduce renal phosphate wasting and alleviate hypophosphatemia [36].