IL17A and endometriosis: As a stimulator of recruitment and activation of immune cells, such as monocytes and neutrophils, upregulation of IL-17 could further activate many downstream cytokines, such as IL-1, IL-6, IL-8, and MCP-1, and participate in the occurrence of endometriosis-related infertility by aggravating inflammation, enhancing toxic effects, and affecting the function of the female reproductive system.