Moreover, hUC-MSC-derived exosomes exhibit antioxidation effects by activating the Nuclear factor erythroid-derived 2-like 2 (Nrf2)/NAD(P)H dehydrogenase, Quinone 1 (NQO-1) signaling pathway, which is crucial for MASH treatment (75). The gene discussed is NFE2L2; the disease is metabolic dysfunction-associated steatohepatitis.