IgA-only sublamina densa LABD, the provisional diagnosis in our patient, is now re-classified as IgA epidermolysis bullosa acquisita (IgA EBA) and demonstrates dermal deposition on SSS, u-serrations on DIF, and collagen VII autoantibody targeting, analogous to classic IgG EBA.1 The gene discussed is CD79A; the disease is acquired epidermolysis bullosa.