Hur et al found that in patients with hyperlipidemia, fat deposits in vessel walls narrow the vessel lumen, increase blood viscosity, and slow blood flow, ultimately leading to deep vein thrombosis.[34] Ay et al suggested that abnormal lipid metabolism in hyperlipidemia inhibits the fibrinolytic system, increases coagulation factors such as thrombin, factor XII, and factor VII, thereby promoting thrombus formation.[35]. The gene discussed is F7; the disease is deep vein thrombosis.