GPR18 and infection: CD206 and CD163are phagocytic receptors and markers of the anti-inflammatory andpro-resolving phenotype, thus indicating that phagocytic receptorsCD163 and CD206 contribute to RvD2-enhanced macrophage phagocytosis.In an acute murine peritonitis model, RvD2 limited PMN infiltrationand enhanced efferocytosis in a GPR18-dependent manner, while GPR18-deficientmice displayed impaired resolution of infections and diminished responseswith RvD2.