BFT binds to a specific colonic epithelial receptor, activating the Wnt and NF-κB signaling pathways, resulting in increased cell proliferation, epithelial release of proinflammatory mediators, and DNA damage (Sears, 2009; Goodwin et al., 2011), whereas ETBF promotes tumor formation in experimental animals (Wu et al., 2009; Goodwin et al., 2011). This evidence concerns the gene NFKB1 and neoplasm.