SRSF2 and myelodysplastic syndrome: Pollyea et al. reviewed the relationship between splicing factor‐mutated MDS and innate immunity contributing to disease pathogenesis, discussing the fact that mutations in SF3B1, U2AF1, and SRSF2 enhance NFκB activity and LPS‐induced inflammatory cytokine production in cell lines, primary patient samples, and murine models.146