While most patients who morphologically have ring‐sideroblasts on bone marrow examination have SF3B1 mutations, outcomes of SF3B1 wild‐type patients with ring‐sideroblasts were more like MDS low blasts than SF3B1‐mutated disease, highlighting the importance of both morphological assessment and molecular testing.35 This evidence concerns the gene SF3B1 and myelodysplastic syndrome.