In contrast, αsyn aggregate interactions (i.e., BAR-PSER129) were markedly different between MSA and PD/DLB, with several glial-enhanced proteins (CBR1, CRYAB, and GFAP) (Human Protein Atlas proteinatlas.org [58]) [7] being more abundant in MSA αsyn aggregates (i.e., PD vs. MSA contrast). The gene discussed is GFAP; the disease is Parkinson disease.