The syndrome of dystonia‐parkinsonism can be acquired (medication‐induced, lesion‐associated, toxic, infectious, prion, autoimmune) and genetic (TAF1, PRKRA, ATP7B, GBA, HTT, SLC19A3, ATP1A3, KMT2B, PLA2G6, GCH1, PRNP, and mitochondrial disorders—many of which are associated with iron accumulation).10 Here, ATP7B is linked to Parkinson disease.