The syndrome of dystonia‐parkinsonism can be acquired (medication‐induced, lesion‐associated, toxic, infectious, prion, autoimmune) and genetic (TAF1, PRKRA, ATP7B, GBA, HTT, SLC19A3, ATP1A3, KMT2B, PLA2G6, GCH1, PRNP, and mitochondrial disorders—many of which are associated with iron accumulation).10 The gene discussed is SLC19A3; the disease is Dystonia.