The syndrome of dystonia‐parkinsonism can be acquired (medication‐induced, lesion‐associated, toxic, infectious, prion, autoimmune) and genetic (TAF1, PRKRA, ATP7B, GBA, HTT, SLC19A3, ATP1A3, KMT2B, PLA2G6, GCH1, PRNP, and mitochondrial disorders—many of which are associated with iron accumulation).10 This evidence concerns the gene SLC19A3 and Parkinsonism.