CASC3 and neoplasm: Althoughthe precise mechanism underlying the enhanced sensitivity of BTZ-resistantMM cells to ATBL remains elusive, we speculate that ATBL achievesbetter results as it does not induce tumor-supporting, host-mediatedeffects shown to occur in response to BTZ in its free form.8 Indeed, we found that ATBL had no effect on MMcell proliferation and migration.