RARRES2 and endometriosis: These results raised three questions: (1) Under normal physiological conditions, whether the embryo expresses and releases chemerin to regulate the EMT of EECs; (2) In obese women of childbearing age, whether overproduced chemerin intervened in the EMT of EECs before the embryo attachment; (3) Overexpression of chemerin was found in some uterine‐related diseases such as endometriosis [40], and whether chemerin affects the lesion tissue invasion through EMT.