A lower prevalence of olfactory deficit among LRRK2-associated parkinsonism has been previously identified,40,51 but in PPMI it has been demonstrated that this finding is largely restricted to LRRK2-associated parkinsonism without evidence of asyn aggregates.4 In a study40 of 530 LRRK2-associated parkinsonism and compared with 759 sporadic Parkinson’s Disease cases, female LRRK2 parkinsonism individuals were less likely to have olfactory deficit.40 However, in that study, biological characterization was not present. The gene discussed is LRRK2; the disease is Parkinson disease.