Interestingly, OSB emissions significantly reduced the release of CCL2 from TCS‐stimulated and unstimulated keratinocytes and slightly reduced IL‐1β from TCS‐stimulated keratinocytes (Figure S8B), confirming no or rather beneficial effects of OSB‐specific VOCs in a human in vitro atopic dermatitis model. Here, CCL2 is linked to atopic eczema.