Moreover, a comprehensive genome-wide sequencing analysis defined a high frequency of Bcor mutations in murine B-cell lymphomas, demonstrating that genetic disruption of Bcor accelerates the MYC-driven lymphomagenesis in the Eμ-Myc mouse model [18], further supporting the tumor suppressor role of Bcor in RT mice. The gene discussed is BCOR; the disease is B-cell non-Hodgkin lymphoma.