We propose a hypothesis that GmZFP2 may target unknown target proteins relying on transcriptional repression activation or direct protein interaction, thus relieving the inhibition of RBOH; alternatively, it may directly suppress the expression of key genes involved in ROS elimination, such as APX, leading to the accumulation of ROS, and further stimulating the plant immune response and augmenting resistance to infections. This evidence concerns the gene APEX1 and infection.