Sickle cell disease (SCD) is an inherited blood condition due to the homozygosity replacing glutamic acid with valine in the β‐subunit of the hemoglobin molecule, causing abnormal hemoglobin variants (Hb S, C, D, E, and O‐Arab and β‐thalassemia) and thereof different forms of SCD [1]. This evidence concerns the gene GSTM1 and Schnyder corneal dystrophy.