Finally, overexpression of Tmem30a also reduced apoptosis marker cleaved caspase‐3 (CC3) and necroptosis marker (p‐MLKL) and increased anti‐apoptosis marker Bcl‐xL and BCL‐2 in the penumbra regions 24 h after stroke (Figure S6D–I). Here, BCL2L1 is linked to stroke disorder.