Moreover, some terms overlapped between the IFNγ-treated and IFNγ-absent conditions, particularly in PD-L1high group, where terms such as peptidyl-lysine modification and covalent chromatin modification indicated the presence of conserved factors involved in tumor-intrinsic PD-L1 regulation, regardless of IFNγ treatment (Fig. 1e). The gene discussed is CD274; the disease is neoplasm.