Hepatitis C infection can lead to the development of STAT1, BUB1, and CDK1, which are known to be involved in the regulation of the cell cycle, oocyte meiosis, human papillomavirus infection, and hepatitis B. Additionally, these proteins have been found to play a role in immune-related pathways, such as interferon signaling and defense responses, which highlights their importance in the host immune response to Dengue virus infection [60]. This evidence concerns the gene CDK1 and hepatitis C virus infection.