The study included participants from New Zealand (925 gout cases and 709 controls) and the Atherosclerosis Risk in Communities (ARIC) study (148 cases and 6927 controls) and indicated that individuals carrying the gout-protective allele in SLC2A9 showed a tendency toward increased effect of fructose consumption upon gout and serum urate elevation (60). This evidence concerns the gene SLC2A9 and gout.