This finding is consistent with in vitro assays showing that high levels of TBXT cause upregulation of markers of epithelial‐to‐mesenchymal transition (EMT) and increase motility and invasiveness of cancer cells, while knocking down TBXT limits tumor metastasis in in vivo models [7, 13], suggesting that TBXT drives an invasive phenotype. The gene discussed is TBXT; the disease is neoplasm.