However, despite the interaction of these motifs with the CoREST complex in B-ALL cells, our MTT assays showed that, unlike the forced expression of full-length ZNF217, forced expression of the ZNF2171-272 truncation failed to rescue the growth inhibition caused by ZNF217 KO (Figure 5D and 5E). The gene discussed is ZNF217; the disease is precursor B-cell acute lymphoblastic leukemia.