LPL and hypertensive disorder: In the current study, the presence of non-HTN latent subgroup that was characterized by dyslipidemia, specifically high serum TG and/or low HDL, with a very low likelihood of hypertension, could be explained through mechanisms related to lipoprotein metabolism and genetic factors that do not necessarily involve hypertension such as hepatic overproduction of VLDL (59), impaired lipoprotein lipase activity or lipoprotein metabolism (60, 61).