In the present study, PR1 and its acidic isoform PR1a1, PR2 (β-1,3-glucanase), PR5 (Thaumatin), PR3 (Chitinase) and PR12 (defensin) were some of the major PR gene transcripts that were majorly induced by pathogen infection/GC pre-treatment or both. Here, PRTN3 is linked to infection.