GHRH and Alzheimer disease: Further pseudo‐time DEG analysis revealed that pathways such as “circadian entrainment,” “glutamatergic synapse,” “axon guidance,” and “giycossphingolipid biosynthesis‐lacto and neolacto series” were upregulated in CGE1, while “cAMP signaling pathway,” “long‐term depression,” “giycossphingolipid biosynthesis‐lacto and neolacto series,” and “pancreatic secretion” pathways were downregulated in CGE2 (Figure S6F,G), linking these two InN subtypes with AD.