ABCG2 and Alzheimer disease: Additional pseudo‐time DEG analysis demonstrated that “adherens junction,” “bile secretion,” “circadian entrainment,” “glutamatergic synapse,” “oxytocin signaling pathway,” “dopaminergic synapse,” and “ABC transporter” pathways were significantly downregulated in Astro1 cells (Figure S7F,G), indicating that astrocyte involvement in AD risk pathways through various regulatory mechanisms.