Furthermore, evidence for the role of MIF in the development, pathogenesis, and treatment of SLE, spondyloarthritis, juvenile idiopathic arthritis, granulomatosis with polyangiitis, Type 1 diabetes, multiple sclerosis, and autoimmune myocarditis have been reported (6, 11, 50–52). The gene discussed is MIF; the disease is autoimmune myocarditis.