In addition, we found that IFN‐γ‐induced BTN3A1 expression in CC cells was dependent on JAK‐STAT1 signaling pathway, which is reminiscent of the mechanism of IFN‐γ‐induced PD‐L1 expression in tumor cells[57] (Figure 8e; Figure S10b, Supporting Information). The gene discussed is CD274; the disease is neoplasm.