To better simulate the in vivo effects of BTN3A1, which is expressed on the cell membrane of tumor cells,[52] we transfected BTN3A1 into K562 cells, which showed very low baseline BTN3A1 expression, to establish BTN3A1‐overexpressed K562 cells (K562‐BTN3A1OE) (Figure S9i, Supporting Information), and used it (Mitomycin C pretreated) to replace BTN3A1‐Fc fusion protein used in above experiments. The gene discussed is BTN3A1; the disease is neoplasm.