For instance, STAT3 has been recognized as a gene associated with dormancy in breast cancer;[51] FOXP1 has been shown to enhance characteristics akin to cancer stem cells in ovarian cancer cells;[52] FOXO3 is known to regulate cell cycle arrest and metabolism;[53] SMADs contributes to tumor dormancy through TGF‐β signaling;[54] SOX9 is responsible for maintaining pluripotency,[55] and facilitating the reprogramming of prostate cancer cells.[56] In addition, the pathway enrichment of cytokine responses (Figure 7J) implies potential intercellular communication via a ligand‐receptor mechanism. This evidence concerns the gene FOXP1 and neoplasm.