While the present results shed no light on differential responses in the development of hypertension in animals early exposed to a hyper-sodium environment, the model has allowed us to characterize other aspects of the phenotype induced by perinatal programming, such as salt consumption and changes in gene expression in the brain (AT1R) and kidney (AT1R, V2R and TRPV1) under a hypertensive challenge. This evidence concerns the gene AGTR1 and hypertensive disorder.