In a STEMI mouse model, researchers observed that LRP1 gene expression and LRP1 protein levels were low in the heart infarction area during the inflammatory reaction stage (day one after STEMI), whereas in the fibrotic stage (days 10 and 21 after STEMI), the expression of LRP1 gene and protein levels in myocardial tissue and fibroblasts and the infarction area were significantly increased. This evidence concerns the gene LRP1 and infarction.