This interaction plays a significant role in the pathophysiology of MALT lymphoma, a finding that aligns with previous research that has reported the expression of CXCL13 and CXCR5 in ocular adnexal MALT lymphoma, suggesting their potential role in mediating lymphocyte homing and retention within the ocular adnexa (38, 39). The gene discussed is CXCR5; the disease is MALT lymphoma.