Despite lacking protein-coding potential, RBPmap [30] analysis identified binding sites for key RNA-binding proteins (RBPs) implicated in GBM (Figure S3B), including IGF2BP, ELAVL4, and MSI1, which regulate processes such as RNA splicing, stability, and tumor cell self-renewal [31,32,33]. The gene discussed is ELAVL4; the disease is neoplasm.