Meanwhile, in the chronic manifestation (post-COVID-19 condition), the main problem is related to the limited regeneration of respiratory cells, as has been previously described [41], characterized by the participation of FAM13A in injury repair and tissue regeneration, DSP in the maintenance of airway epithelial integrity, TERT mediating cell proliferation, and the promotion of fibrotic mechanisms of THSD4 via the TGF-β pathway. The gene discussed is FAM13A; the disease is COVID-19.