We repeated the PGEE analysis with this dataset and confirmed the gene signatures that previously emerged in the first PGEE analyses, except for ABHD5, RBM38, and IFI16. Additional genes emerged with significantly different expression in the GVHD versus non-GVHD cohorts: CDC34, TRAK2, SERF2, belonging to “erythroid” modules; GPB1 and STAT1 belonging to “IFN” modules; and the single genes MSN (neutrophils), GPSM3 (inflammation), TLR2 (cytokines/chemokines), and PFAAP5 (protein synthesis). This evidence concerns the gene IFNA1 and graft versus host disease.