“Erythroid”-, “inflammation”-, “neutrophil/neutrophil activation”-, and “protein synthesis”-related gene signatures, as well as the TLR2 gene, also distinguished GVHD traits without infection/relapse interference, along with modules M10.4 “neutrophil activation”, M12.15 “cell cycle”, and 13.16 “protein synthesis”. The gene discussed is TLR2; the disease is graft versus host disease.