NTRK1 and Cirrhosis: Cirrhosis-free patients with early recurrence exhibit impaired FGFR signaling, the PI3K network, the RUNX1/3-regulated pathway, the MAPK pathway, and tyrosine kinase receptor signaling, and elastic fiber formation and laminin interactions contributed to the epithelial–mesenchymal transition (EMT) in the progress of metastasis.