ATM and idiopathic pulmonary fibrosis: The important findings could be summarized as follows: By biological information analysis, it was found that ATM was the hub gene related to ferroptosis that co-expressed in the serum and pulmonary tissue of patients with IPF; ELISA verification revealed that the concentration of ATM in the serum of patients with IPF significantly decreased; IHC verification revealed that the expression level of ATM in pulmonary tissue of patients with IPF was significantly increased; there was a significant correlation between ATM and immune-infiltrating cells in serum and pulmonary tissue.