Glanzmann’s thrombasthenia (GT) is a rare genetic autosomal recessive bleeding disorder characterized by a deficiency or functional defect in the platelet surface glycoprotein (GP) IIb-IIIa receptor complex, also known as the CD41-CD61 receptor complex located on the platelet surface [1–4] due to genetic mutations in the ITGA2B and ITGB3 genes that are situated adjacently on region two of the long arm of chromosomes 17 [5–7]. This evidence concerns the gene ITGA2B and Glanzmann thrombasthenia 1.