FOXP3 and uveitis: Interestingly, IL-17 has been implicated in the induction of Foxp3+ Treg cells within the EAU model [16], suggesting a complex interplay where IL-17 and Th17 response can contribute to both the onset and the mitigation of ocular inflammation, which may explain the inconsistent results of IL-17 neutralizing agents in treating uveitis [10].