According to their research, ferulic acid reduced ROS overproduction, promoted GSH production, blocked Ptgs2 (prostaglandin-endoperoxide synthase 2) mRNA and Fe2+ accumulation, decreased LDH (lactate dehydrogenase) and CK (creatine kinase) activities, attenuated myocardial infarction, and improved ischemia–reperfusion-induced ferroptosis. The gene discussed is PTGS2; the disease is myocardial infarction.