MTOR and acute myeloid leukemia: According to Du et al,109 dihydroartemisinin accelerates ferritin degradation, increases the unstable iron pool, encourages the formation of ROS in cells, and ultimately causes ferroptosis in acute myeloid leukemia cells by controlling the activity of the AMPK/mTOR (mammalian target of rapamycin)/p70S6k signaling pathway.109