Previous studies have demonstrated that MGAT4EP enhances FOXA1 binding to its promoter in luminal A tumors, leading to the upregulation of FOXM1 and promoting breast cancer growth and metastasis.18 To explore the biological functions of MGAT4EPmore thoroughly, we performed GSEA analysis and identified that MGAT4EP might regulate multiple key signaling pathways involved in breast cancer development, particularly those related to apoptosis and cell adhesion. The gene discussed is FOXA1; the disease is breast carcinoma.