The results indicated that the combined effect of LUT and AA involved the inactivation and activation of multiple signaling targets, and induced apoptosis by downregulating PI3K/AKT (PI3K, AKT and p70S6K) and JNK/p38 MAPK signaling pathways, upregulating the ERK signaling pathway, resulting in an anticancer effect on cervical cancer. This evidence concerns the gene RPS6KB1 and cervical carcinoma.