Additionally, the expression of MyD88, NF-κB p65, iNOS, and Arg-1 was significantly elevated in the E. granulosus group (Figures 7C–F), confirming that GHSR-/- mice exhibited milder infection and a lower inflammatory status, further supporting the conclusion that GHSR deficiency alleviates E. granulosus infection. Here, GHSR is linked to infection.