YTHDF2 and hepatocellular carcinoma: Studies have shown that YTHDF2 undergoes O‐GlcNAcylation after HBV infection, increasing its stability and oncogenic activity, which promotes the cell cycle by stabilizing m6A‐modified transcripts like minichromosome maintenance 2 (MCM2) and MCM5, further driving HBV‐related HCC [94].