This research investigates the role of inhibitor of differentiation 2 (Id2) in the synthesis of pro‐inflammatory cytokines, specifically interferon‐γ (IFN‐γ) and interleukin‐17 (IL‐17), by various subsets of T cells, and its pathogenic role in rheumatoid arthritis (RA). Here, IL17A is linked to rheumatoid arthritis.