Interestingly, and similar to the situation in NEO versus AD Mo, E2F1 and FLI1 were oppositely (Fig. 7d, m) and STAT1 and FLI1 concordantly (Fig. 7j, m) differentially expressed in their high versus low donor subgroups, pointing again to contrary (E2F1 versus FLI1) respective similar (STAT1 and FLI1) regulatory functions. The gene discussed is STAT1; the disease is Alzheimer disease.