SLC2A1 and Parkinson disease: Considering the key role of EAATs in Glut transport as well as the fact that they are expressed at both plasma membrane and mitochondrial levels [21, 58] of particular interest is our observation that silencing of EAAT3, but not the inhibition of plasma membrane EAATs with TFB‐TBOA, fully prevented the BCH‐induced neuroprotection in our PD model.