The stability of the lncRNA DIAPH1-AS1 can also be improved by m6A modification mediated by WTAP through a pathway dependent on IGF2BP2, whereupon it functions as a molecular adaptor capable of promoting the formation of the MTDH-LASP1 complex and upregulating LASP1 to facilitate the growth and metastasis of nasopharyngeal carcinoma [51]. Here, LASP1 is linked to nasopharyngeal carcinoma.