This scenario may also apply to the immunopathogenesis of human type 1 AIP because serum IFN-α, CCL25, CXCL9, and CXCL10 concentrations are significantly higher in patients with type 1 AIP than in healthy controls and those with chronic alcoholic pancreatitis. Here, CXCL9 is linked to autoimmune pancreatitis type 1.